Qin Li's research while affiliated with Southern Medical University and other places
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Publications (3)
Background
Long non-coding RNA (lncRNA) dysregulation is demonstrated to be associated with disease progression. Mounting studies show that lncRNA promotes or inhibits the development of keloid. We aimed to disclose the role of homebox A11 antisense RNA (HOXA11-AS) in the formation of keloid.
Methods
Quantitative real-time PCR (qPCR) was adopted f...
A diabetic nonhealing wound causes heavy economic burden and compromised quality of life in patients. The human dermal fibroblast (HDF), which is an important kind of effector cell in the wound healing process, represents different biological behaviors in the normal and diabetic skins. Given this, we attempt to explore functional changes in diabeti...
Citations
... Furthermore, ZNF217 is a downstream target of miR-182-5p. 84 In keloid fibroblasts, long non-coding RNA HOXA11-AS indirectly regulates the expression of ZNF217 by competitively binding to miR-182-5p, which in turn controls the formation and development of keloids 84 (Figure 4). ...
... After being released from donor cells, exosomes are distributed in biological fluids and taken up by the same or different types of cells to exert biological regulatory function (Sahoo and Losordo, 2014). Exosome microRNAs (miRNAs), the main molecules playing a regulatory role in exosomes, primarily cause gene silencing in receptor cells (Sato et al., 2017a;Liu et al., 2021). The transfer of exosome miRNAs between DCs is considered a means of communication and posttranscriptional regulation (Montecalvo et al., 2012). ...
... Another miRNA, miR1273p, regulates the transition from the proliferative phase to the remodelling phase of mouse wounds, possibly by inhibiting the proliferation of myofibroblasts 155 . In human wound healing, miR1273p might function as an epigenetic activator that regulates the transition from repair to remodelling during skin wound healing 155,156 . Finally, miR210 is induced by tis sue hypoxia and targets keratinocyte proliferation 157 , and inhibition of miR210 is being investigated as a way to increase keratinocyte proliferation and accelerate heal ing. ...
Reference: Chronic wounds
