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Rutherford Vascular Surgery 6th Edition, Elsevier Saunders (2005) 2736 pages, £210.00

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Book Review
Rutherford Vascular Surgery 6th Edition,Elsevier
Saunders 2005, 2736 pages, £210.00.
The new Edition of Vascular surgery with 2736 pages
has expanded substantially and is recently updated
with several new chapters. The book is divided into
23 chapters with overwhelmingly North-American
contributors. Several of the chapters are completely
re-written by new authors, whereas others are up-
dates of previous chapters.
The particular strength of this book, and also being
the first of its kind, is that in the online version, there
are regular updates retrievable through the world-
wide web, which makes the book outstanding in its
field. For those who would look for updates in a spe-
cific field and who prefer to get ‘‘filtered’’ or
previewed information this is possible to get through
these updates, which are like a condensed abstract,
resulting in a good opportunity to get such
information.
The present edition is like previous ones, systemat-
ically dealing with basic considerations diagnostic
tools, physiology as well as the non-operative man-
agement of patients with vascular diseases. These
chapters are very well written and also with necessary
updates resulting in a book with a very short time-
delay between the appearance of original publications
and the manifestation in the textbook, which is not
always the case in medical textbooks.
The chapter on the generalised arteriosclerosis of
the vascular patient is extensively described also
presenting a pathway for drug-treatment, helping
the vascular surgeon without an angiologist on his
side to effectively treat his patient. The chapter on
vascular pain is very useful too, especially for youn-
ger colleagues trying to correlate the patient’s symp-
toms with clinical findings. The chapter on
abdominal aortic and iliac aneurysms is very well
written with fresh updates of all the latest rando-
mised trials and dealing with such important topics
like decision making when to operatively treat an
elective aortic aneurysm. The complete chapter on
the endovascular treatment of aortic aneurysms is
also well written including coverage of various stent
grafts.
‘‘Fundamentals in endovascular surgery’’ has got
its own chapter and is very constructive for those vas-
cular surgeons, who have not yet entered the endo-
vascular arena.
The results of such endovascular techniques, in-
cluding thrombolytic therapy are well addressed
in the chapters on acute limb ischemia, and intra-
arterially catheter directed thrombolysis.
The chapter on venous malformation is too very
well written and divided in such a way that it is
easy to specifically find answers to narrow questions
and rarely appearing diseases.
The chapter on vascular thoracic trauma, though, is
almost only dealing with open techniques in the arse-
nal for managing these injuries, and very little is high-
lighted on the new endovascular management, which
at least in the present time in Europe, has been shown
to produce good results. It would have been prefer-
able to have this chapter being expanded with the
results using these modalities.
The chapter on venous access and its complications
is well written but I am missing some of the unusually
shunts like subclavian to subclavian or subclavian to
jugular just to mention a few. This is especially impor-
tant since venous access is becoming increasingly
important for the vascular surgeon.
This bible in Vascular Surgery should be in the
library of every vascular department and it is of
a great value for a fulltime vascular and endovascular
surgeon, but could be used by general surgeons who
want to be updated in the vascular field. This book is
highly recommended, and with all its figures and il-
lustrations it is easy to read and can also be used for
students with specific or special interest of vascular
surgery.
J. Brunkwall
Department of Vascular Surgery,
University of Cologne,
Joseph Stelzmannstrasse 9,
D-50931 Cologne, NRW, Germany
E-mail address: jan.brunkwall@uk-koeln.de
Accepted 23 April 2006
Available online 13 June 2006
Eur J Vasc Endovasc Surg 32, 337 (2006)
doi:10.1016/j.ejvs.2006.04.025, available online at http://www.sciencedirect.com on
1078–5884/000337 + 01 $35.00/0 Ó2006 Published by Elsevier Ltd.
Chapter
Development of the vascular system involves continual growth and regression of vascular structures. Thus, each segment of the aorta is subject to variations in anatomy. The main limitation for endovascular repair of aortic pathology has been the need to maintain perfusion to the arch, mesenteric, renal, and iliac branches of the aorta. To properly plan and treat aortic segments spanning these branch zones, understanding their anatomic relationships and common variations in arterial anatomy is critical. This chapter focuses on normal and common variations of the aorta and its major branches that have implications in the use of fenestrated and branched endografts.
Chapter
Managing a patient with a vascular surgical emergency can be challenging. Often a patient will have multiple comorbidities, an extensive surgical history, and a lengthy medication list including novel anticoagulation and antiplatelet agents. Even if managed in a timely fashion, vascular emergencies can lead to significant and long-term morbidity or mortality.
Chapter
This chapter functions as a discussion and step-by-step illustrated guide to the common vascular exposures from the aorta to the peripheral vasculature that a general surgeon may need to call upon during emergencies or planned operations. The exposures themselves are tools, but this chapter also includes an introduction to the commonly used instruments and sutures as well as a guide to balloon sizing. Varying approaches to anastomoses and means of obtaining vascular control are also discussed. Vascular surgery in the twenty-first century requires a basic understanding of angiography. The option to perform on table imaging, preoperative imaging for planning, or to offer a hybrid interventional approach is unique to the current era of vascular surgery. For the nonvascular trained surgeon, angiography can be a daunting task so herein the basics of angiography and maneuvers to ensure safety during radiation exposure are also discussed.
Chapter
The prevalence of atherosclerotic renal artery stenosis has been described in population-based, autopsy, and referred populations and has been summarized in multiple reviews. The prevalence is highest among patients with known atherosclerotic disease. For example, in 319 patients undergoing catheter-based angiography for aortic or peripheral atherosclerotic disease, >50 % stenosis of a renal artery was found in 38 % patients. The prevalence remains high for patients suspected to have coronary artery disease. In over 20,000 patients referred for coronary angiography across seven studies, the prevalence of >50 % renal artery stenosis by angiography ranged from 6.3 to 23 %. In the general population, the prevalence is lower but not insignificant. That is, in a study of 834 Medicare patients with a mean age of 77 years, nearly 7 % had a renal artery stenosis of >60 % by screening duplex ultrasonography.
Article
Full-text available
Buerger’s disease (BD) or thromboangiitis obliterans is a vasculitis that most commonly affects the small and medium-sized arteries and veins in the extremities.1 It is most frequently seen in the young men who smoke and is associated with low socioeconomic status. BD is diagnosed on the basis of the clinical findings; the pathogenesis is not completely be understood.1-4 In this report, we aim to present the clinical, magnetic resonance imaging (MRI) and angiographic findings of a 30-year-old man with ischemic stroke as a rare complication of BD.
Article
OBJECTIVE: Post-thrombotic syndrome (PTS) is the long-term sequelae of deep venous thrombosis (DVT). PTS clinical manifestations include chronic leg pain, oedema, lipodermatosclerosis and ulcers. The objective of this study is to determine in patients with documented history of thrombophilias and DVT whether the number of previous thrombotic events and optimal anticoagulation therapy are associated with the time to venous ulcer healing following the start of compression therapy. METHOD: Retrospective analysis performed in thrombophilic patients under the age of 50 years old with chronic venous ulcers secondary to DVT at the wound clinic in the National Institute of Medical Sciences and Nutrition 'Salvador Zubirán ' in Mexico City. Variables such as the number or episodes of thrombotic events, type of hypercoagulable disorder, optimal anticoagulation therapy with Warfarin monitored by therapeutic International Normalised Ratio (INR) (2-3) and compliance to compression therapy were examined. Patients that underwent superficial or perforator vein interruption or endovascular recanalisation of deep veins were excluded from the study. RESULTS: From a database of 29 patients with chronic venous ulcers followed in our clinic from January 1992 to September 2012, only 13 patients (61% female) met the inclusion criteria. Mean age±standard deviation (SD) was 32±12 years old. Of these, seven (54%) patients with suboptimal INR presented with an average of two previous thrombotic events and the remaining six (46%) patients with optimal INR only one event (p=0.28), the mean time to the clinical manifestation of a venous ulcer after the first episode of DVT was 39 months (range: 12-72) for patients with suboptimal INR and 82 months (range: 12-216) for those with optimal anticoagulation therapy (p=0.11). During the mean follow-up period of 52 months, all patients in optimal anticoagulation healed their ulcer; their mean time for wound healing was 44 months (range: 4-102). In the suboptimal INR group, only four healed the ulcers with an mean of 72 months (range: 2-204) (p=0.94). CONCLUSION: There seems to be an association between an optimal anticoagulation therapy with Warfarin monitored by INR and wound healing rates in thrombophilic patients with chronic venous ulcers. Further research is warranted.
Chapter
Due to dramatic advances in endovascular techinques, multiple approaches to the treatment of chronic limb ischemia (CLI) are widely available to a variety of specialists. Although most of these techinques are often succesful in restoring flow to the ischemic extremity, it is unclear if this immediate success translates into long-lasting wound healing, limb salvage and retrun to a pr-morbid level of activity.Traditionally, the “success” of vascular interventions has been measured solely in terms of immediate technical success, patency, freedom from further revascularization, limb salvage and mortality. Outcomes research in CLI is entering a new phase where patient-oriented outcomes are replacing traditional lesion-oriented outcomes.
Chapter
Acute limb ischemia (ALI) is defined as any sudden decrease in limb perfusion causing a potential threat to limb viability (Norgren et al., J Vasc Surg 45:S5–67, 2007). The incidence of ALI is 9–16 cases per 100,000 persons per year for the lower extremity (Creager et al., N Engl J Med 366:2198–2206, 2012; Dryjski and Swedenborg, J Cardiovasc Surg 25:518–522, 1984; Davies et al., Br J Surg 84:504–508, 1997) and around 1–3 cases per 100,000 persons per year for the upper extremity (Eyers and Earnshaw, Br J Surg 85:1340–1346, 1998). Etiology includes embolism, in situ thrombosis with coexisting peripheral arterial disease (PAD), graft/stent thrombosis, trauma, or peripheral aneurysm with embolism or thrombosis. ALI management makes up 10–16 % of the vascular workload for the average vascular specialist. Amputation and mortality rates are historically high in these patients, however, with advances in anticoagulation and surgical therapy that have decreased over time.
Article
Full-text available
Background: Osteochondroma is the most common non-malignant tumour of bone, accounting for approximately one third of benign lesions in the skeleton. They often develop around the knee in the distal femur and in the proximal tibia and fibula. They present as a painless slow growing mass during adolescence and have been reported to cause damage to adjacent structures such as blood vessels; arterial damage is more common than venous injury and is usually a result of compression, stretching, and rubbing of the arterial wall. Such lesions include stenosis, thrombosis, and pseudoaneurysm formation possibly causing lower limb claudication or acute limb ischemia. Methods: An 18 year old male patient with a 4 week history of pain, hematoma, and oedema of the left calf without previous trauma is reported. A computed tomography scan (CT) revealed a large popliteal artery pseudoaneurysm and its close relationship to a protrusion of the proximal tibia. Results: The popliteal artery was repaired by an external saphenous patch and the exostosis was removed. The patient had palpable popliteal and distal pulses after surgery and during the first year follow-up. Conclusions: Tibial osteochondroma should be considered in the differential diagnosis in young patients, among the potential causes of pseudoaneurysm of the femoral or popliteal artery. Surgical repair should be performed to restore normal blood flow with resection of the exostosis to prevent recurrence.
Article
A 42-year-old man had intestinal ischemia 7 weeks after endovascular abdominal aortic aneurysm repair due to sacrifice of the inferior mesenteric artery, which had compensated for the intestinal blood supply because of the total occlusion of the superior mesenteric artery (SMA) and severe stenosis of the celiac artery (CA). He was diagnosed in the active phase of Takayasu arteritis, and an emergency endovascular treatment was performed. After the SMA failed to be recanalized, a stent was successfully placed into the CA; this choice was made based on the preexisting collaterals between them. The symptoms were relieved shortly after the operation. The Kirk arcade, the Barkow arcade, and the enlarged pancreaticoduodenal arcade were visualized on the follow-up computed tomography angiography. Based on this case, a short review of celiomesenteric and intermesenteric collateral circulations is presented.
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