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1­ Reactive oxygen species (ROS) formed under high salt concentration due to ionic imbalance results in oxidative stress and damage to lipids, proteins, nucleic acids, and carbohydrates. Source: Sharma et al. (2012). Licenced under CC BY-3.0.

1­ Reactive oxygen species (ROS) formed under high salt concentration due to ionic imbalance results in oxidative stress and damage to lipids, proteins, nucleic acids, and carbohydrates. Source: Sharma et al. (2012). Licenced under CC BY-3.0.

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Salinity is one of the important menaces to crop plants. Most of the plants including agricultural crops are glycophytic and are unable to complete their life cycle in high concentration of salt. This chapter summarizes the consequences of salinity on crop plants, oxidative biomarkers, and their effects in plants under salt stress, and discusses th...

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... ROS levels can disrupt biomolecules including lipids, proteins, carbohydrates, and DNA. Such reactions can modify intrinsic membrane properties such as fluidity, transport of ions, loss of enzyme activity, cross-linking of proteins, inhibition of protein synthesis, harm to DNA, and so on and eventually result in plant cell death (Figure 9.1). ...
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... seed germination, root length, plant height, and fruiting of plants are significantly inhibited under salt treatments ( Liang et al. 2014). Plants adopt mainly two strategies to counteract the effects of high salt concentration, namely enzymatic and nonenzymatic strategies (Figure 9.2). ...
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... plants maintain their own ion homeostasis by ion uptake and compartmentalization, which do not play an important role only in normal growth but also essential for growth during salt stress (Hasegawa 2013). The signaling pathway of SOS (salt overly sensitive) having three major proteins, SOS1, SOS2, and SOS3, is shown in Figure 9.3. Out of these three proteins, SOS1 codifies an antiporter Na + /H + plasma membrane and is essential for cellular-level regulation of Na + efflux. ...
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... protein SOS3 then interacts with it and activates the protein SOS2 by triggering its selfinhibition. The complex SOS3-SOS2 is then charged to the plasma membrane where it phosphorylates SOS1 (Figure 9.3). The phosphorylated SOS1 results in increased efflux of Na + , which reduces salt toxicity ( Martínez-Atienza et al. 2007). ...
Context 5
... ROS levels can disrupt biomolecules including lipids, proteins, carbohydrates, and DNA. Such reactions can modify intrinsic membrane properties such as fluidity, transport of ions, loss of enzyme activity, cross-linking of proteins, inhibition of protein synthesis, harm to DNA, and so on and eventually result in plant cell death (Figure 9.1). ...
Context 6
... seed germination, root length, plant height, and fruiting of plants are significantly inhibited under salt treatments ( Liang et al. 2014). Plants adopt mainly two strategies to counteract the effects of high salt concentration, namely enzymatic and nonenzymatic strategies (Figure 9.2). ...
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... plants maintain their own ion homeostasis by ion uptake and compartmentalization, which do not play an important role only in normal growth but also essential for growth during salt stress (Hasegawa 2013). The signaling pathway of SOS (salt overly sensitive) having three major proteins, SOS1, SOS2, and SOS3, is shown in Figure 9.3. Out of these three proteins, SOS1 codifies an antiporter Na + /H + plasma membrane and is essential for cellular-level regulation of Na + efflux. ...
Context 8
... protein SOS3 then interacts with it and activates the protein SOS2 by triggering its selfinhibition. The complex SOS3-SOS2 is then charged to the plasma membrane where it phosphorylates SOS1 (Figure 9.3). The phosphorylated SOS1 results in increased efflux of Na + , which reduces salt toxicity ( Martínez-Atienza et al. 2007). ...
Context 9
... ROS levels can disrupt biomolecules including lipids, proteins, carbohydrates, and DNA. Such reactions can modify intrinsic membrane properties such as fluidity, transport of ions, loss of enzyme activity, cross-linking of proteins, inhibition of protein synthesis, harm to DNA, and so on and eventually result in plant cell death (Figure 9.1). ...
Context 10
... seed germination, root length, plant height, and fruiting of plants are significantly inhibited under salt treatments ( Liang et al. 2014). Plants adopt mainly two strategies to counteract the effects of high salt concentration, namely enzymatic and nonenzymatic strategies (Figure 9.2). ...
Context 11
... plants maintain their own ion homeostasis by ion uptake and compartmentalization, which do not play an important role only in normal growth but also essential for growth during salt stress (Hasegawa 2013). The signaling pathway of SOS (salt overly sensitive) having three major proteins, SOS1, SOS2, and SOS3, is shown in Figure 9.3. Out of these three proteins, SOS1 codifies an antiporter Na + /H + plasma membrane and is essential for cellular-level regulation of Na + efflux. ...
Context 12
... protein SOS3 then interacts with it and activates the protein SOS2 by triggering its selfinhibition. The complex SOS3-SOS2 is then charged to the plasma membrane where it phosphorylates SOS1 (Figure 9.3). The phosphorylated SOS1 results in increased efflux of Na + , which reduces salt toxicity ( Martínez-Atienza et al. 2007). ...