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Vias de aumento de intermediários do ciclo de Krebs (anaplerose). PEPCK, fosfoenol piruvato quinase; PC, piruvato carboxilase; EM, enzima málica; CS, citrato sintase; ISCD, isocitrato desidrogenase; -CGTD, -cetoglutarato desidrogenase; S- CoA sintase; succinil-CoA sintase; SCD, succinato desidrogenase; MLD, malato desidrogenase; AMP, adenosina monofosfato; CPN, ciclo das purinas nucleotídios; BCAA, aminoácidos de cadeia ramificada..  

Vias de aumento de intermediários do ciclo de Krebs (anaplerose). PEPCK, fosfoenol piruvato quinase; PC, piruvato carboxilase; EM, enzima málica; CS, citrato sintase; ISCD, isocitrato desidrogenase; -CGTD, -cetoglutarato desidrogenase; S- CoA sintase; succinil-CoA sintase; SCD, succinato desidrogenase; MLD, malato desidrogenase; AMP, adenosina monofosfato; CPN, ciclo das purinas nucleotídios; BCAA, aminoácidos de cadeia ramificada..  

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Figura 1. Efeito do sistema glutationa/tiorredoxina peroxidase na...
Figura 2. Efeito da glicose plasmática e de glutationa hepático na...
Figura 4. Efeito do ciclo glicose-ácido graxo na redução do metabolismo...
Figura 5. Efeito inibitório das Espécies reativas de oxigênio na...
+2 Figura 6. Vias de aumento de intermediários do ciclo de Krebs...
Metabolic regulation and production of oxygen reactive species during muscule contraction: effect of glycogen on intracellular redox state
Article
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  • Jan 2008
Fatigue is closely related to the depletion of glycogen in the skeletal muscle during prolonged exercise. Under this condition, the production of oxygen reactive species (ROS) is substantially increased. It has been shown that dietary supplementation of carbohydrate or antioxidant attenuates muscle fatigue during contraction. This suggests that gly...
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